Annual Neurologists, Neurosurgeons and Medical Practitioners Meet October 21-22, 2019 Vancouver, Canada

Biography:

Respectfully stated, I am a hybrid applicant of both Social Sciences and Medical sciences backgrounds. I earned my master degree in English Linguistics from The University of Hassan II in Morocco. As a master of science student at the Medical University of Vienna and The Center for Brain Research in Vienna, I participated in neuroscience, clinical neuroscience, cognitive neuroscience and cognitive psychology researches. I spent some time in a postgraduate research centre in cognitive neuroscience in Zagreb in Croatia. I had the privilege of doing research in the hospital of Charité, Berlin in German and witness the procedures and methods like polysomnography which have been applied practically on patients with different sleep disorders. At the moment, I am conducting my research as an MSc student at the Medical University of Vienna investigating and profounding my clinical knowledge of sleep disorders in smokers. I am a coauthor of the second eddition of “Alcohol and Tobacco Medical and Sociological Aspects of Use, Abuse and Addiction” in press.

Abstract:

Correlation between smoking and sleep interruptions have been documented in many research papers. Misalignment in the mechanisms of the suprachiasmatic nucleus (SCN), the hypothalamic– pituitary adrenal axis (HPA), or hypothalamus interferes negatively in producing sleep disorders. Earlier animal studies showed that nicotine influences the circadian system via stimulating the nicotinic acetylcholine receptors (nAChRs) in SCN of the hypothalamus. The stimulation of nicotine to the HPA axis leads to the secretion of corticotropin-releasing hormone (CRH) in the paraventricular nucleus of the hypothalamus. This secretion stimulates the production of adrenocorticotropic hormone (ACTH) in the pituitary gland and hence leads to the increased secretion of cortisol by the adrenal gland. Sleep neurotransmitter like GABA and melatonin can be suppressed either through the inhibition of nicotine to neurons in SCN or through the stimulation of nicotine to the HPA functions by increasing the secretion of serotonin, dopamine, and noradrenaline. The purpose of this study is to investigate the relationship of tobacco and sleep disorders in Lesch typology, which is a clinical classification of four types of addicted patients to tobacco based on their level of addiction and clinical severities. Building on our previous data, we assume to see insomnia disorder in type 1 smokers who may have less total sleep time (TST), longer sleep latency, high REM sleep density, in comparison with type 2 and the control group. In this investigation, the Lesch European Smoker Classification instrument and test of biomarkers will be used to assess the tobacco abuse. To assess the sleep structure we aim to use actigraphy in combination with sleep log and polysomnography (PSG).

Biography:

Giovanni Alano Vista graduated from Cebu Institute of Medicine at the age of 25. Presently, he is a second year resident in the Department of Internal Medicine in Perpetual Succour Hospital. He received several awards and distinctions in the field (1) First place during the 11th Annual Intradepartmental Research Contest, October 2018 (2) Third place during the Second Philippine College of Physicians Research Contest, October 2018, (3) second during the Annual Cebu Institute of Medicine Research Contest, December 2018. He was recently offered a position of  Research Subinvestigator for Perpetual Succour Hospital Cebu Cancer Institute.

Abstract:

Spontaneous acute subdural hematoma (ASDH) comprises 2.6% of all ASDH. In one recent study, only 178 cases of spontaneous ASDH were documented, however, only one of them was attributed to the presence of dural arteriovenous fistula (dAVF). Only less than 10% of all subarachnoid hemorrhage (SAH) are caused by vascular malformations. Spontaneous ASDH and SAH occurring together is extremely rare. There is a scarcity of literature on cases with dAVF of the occipital lobe as a cause of simultaneous spontaneous ASDH and SAH.

A 44-year old Filipino male with no history of trauma, presented with severe headache, vomiting, and decreasing sensorium – CT scan revealed acute parenchymal bleed in the left occipital lobe with subarachnoid extension and subdural hematoma in the left frontoparietotemporal convexity along the tentorium cerebeli and posterior interhemispheric falx. Due to the location of the lesion seen on CT scan and the gender distribution, Arteriovenous malformation was initially considered, thus proceeded to CTA to establish the diagnosis of vascular anomaly, however, revealed dAVF instead. Four-vessel angiogram was done to assess the tributaries of the dAVF and confirmed the diagnosis. Complete obliteration of dAVF of the occipital lobe was done with Onyx Embolization in one session.

This is the first case of Borden type II, Cognard type IIa+IIb  dAVF, as reported in this institution.  Although rare as a cause of SAH and ASDH, dAVF should be considered as a differential in patients with the absence of identifiable common cause of new onset of severe headache and poor neurologic status.

Biography:

Respectfully stated, I am a hybrid applicant of both Social Sciences and Medical sciences backgrounds. I earned my master degree in English Linguistics from The University of Hassan II in Morocco. As a master of science student at the Medical University of Vienna and The Center for Brain Research in Vienna, I participated in neuroscience, clinical neuroscience, cognitive neuroscience and cognitive psychology researches. I spent some time in a postgraduate research centre in cognitive neuroscience in Zagreb in Croatia. I had the privilege of doing research in the hospital of Charité, Berlin in German and witness the procedures and methods like polysomnography which have been applied practically on patients with different sleep disorders. At the moment, I am conducting my research as an MSc student at the Medical University of Vienna investigating and profounding my clinical knowledge of sleep disorders in smokers. I am a coauthor of the second eddition of “Alcohol and Tobacco Medical and Sociological Aspects of Use, Abuse and Addiction” in press.

Abstract:

Correlation between smoking and sleep interruptions have been documented in many research papers. Misalignment in the mechanisms of the suprachiasmatic nucleus (SCN), the hypothalamic– pituitary adrenal axis (HPA), or hypothalamus interferes negatively in producing sleep disorders. Earlier animal studies showed that nicotine influences the circadian system via stimulating the nicotinic acetylcholine receptors (nAChRs) in SCN of the hypothalamus. The stimulation of nicotine to the HPA axis leads to the secretion of corticotropin-releasing hormone (CRH) in the paraventricular nucleus of the hypothalamus. This secretion stimulates the production of adrenocorticotropic hormone (ACTH) in the pituitary gland and hence leads to the increased secretion of cortisol by the adrenal gland. Sleep neurotransmitter like GABA and melatonin can be suppressed either through the inhibition of nicotine to neurons in SCN or through the stimulation of nicotine to the HPA functions by increasing the secretion of serotonin, dopamine, and noradrenaline. The purpose of this study is to investigate the relationship of tobacco and sleep disorders in Lesch typology, which is a clinical classification of four types of addicted patients to tobacco based on their level of addiction and clinical severities. Building on our previous data, we assume to see insomnia disorder in type 1 smokers who may have less total sleep time (TST), longer sleep latency, high REM sleep density, in comparison with type 2 and the control group. In this investigation, the Lesch European Smoker Classification instrument and test of biomarkers will be used to assess the tobacco abuse. To assess the sleep structure we aim to use actigraphy in combination with sleep log and polysomnography (PSG).

Biography:

Giovanni Alano Vista graduated from Cebu Institute of Medicine at the age of 25. Presently, he is a second year resident in the Department of Internal Medicine in Perpetual Succour Hospital. He received several awards and distinctions in the field (1) First place during the 11th Annual Intradepartmental Research Contest, October 2018 (2) Third place during the Second Philippine College of Physicians Research Contest, October 2018, (3) second during the Annual Cebu Institute of Medicine Research Contest, December 2018. He was recently offered a position of  Research Subinvestigator for Perpetual Succour Hospital Cebu Cancer Institute.

 

Abstract:

Spontaneous acute subdural hematoma (ASDH) comprises 2.6% of all ASDH. In one recent study, only 178 cases of spontaneous ASDH were documented, however, only one of them was attributed to the presence of dural arteriovenous fistula (dAVF). Only less than 10% of all subarachnoid hemorrhage (SAH) are caused by vascular malformations. Spontaneous ASDH and SAH occurring together is extremely rare. There is a scarcity of literature on cases with dAVF of the occipital lobe as a cause of simultaneous spontaneous ASDH and SAH.

A 44-year old Filipino male with no history of trauma, presented with severe headache, vomiting, and decreasing sensorium – CT scan revealed acute parenchymal bleed in the left occipital lobe with subarachnoid extension and subdural hematoma in the left frontoparietotemporal convexity along the tentorium cerebeli and posterior interhemispheric falx. Due to the location of the lesion seen on CT scan and the gender distribution, Arteriovenous malformation was initially considered, thus proceeded to CTA to establish the diagnosis of vascular anomaly, however, revealed dAVF instead. Four-vessel angiogram was done to assess the tributaries of the dAVF and confirmed the diagnosis. Complete obliteration of dAVF of the occipital lobe was done with Onyx Embolization in one session.

This is the first case of Borden type II, Cognard type IIa+IIb  dAVF, as reported in this institution.  Although rare as a cause of SAH and ASDH, dAVF should be considered as a differential in patients with the absence of identifiable common cause of new onset of severe headache and poor neurologic status.

Biography:

Respectfully stated, I am a hybrid applicant of both Social Sciences and Medical sciences backgrounds. I earned my master degree in English Linguistics from The University of Hassan II in Morocco. As a master of science student at the Medical University of Vienna and The Center for Brain Research in Vienna, I participated in neuroscience, clinical neuroscience, cognitive neuroscience and cognitive psychology researches. I spent some time in a postgraduate research centre in cognitive neuroscience in Zagreb in Croatia. I had the privilege of doing research in the hospital of Charité, Berlin in German and witness the procedures and methods like polysomnography which have been applied practically on patients with different sleep disorders. At the moment, I am conducting my research as an MSc student at the Medical University of Vienna investigating and profounding my clinical knowledge of sleep disorders in smokers. I am a coauthor of the second eddition of “Alcohol and Tobacco Medical and Sociological Aspects of Use, Abuse and Addiction” in press.

Abstract:

Correlation between smoking and sleep interruptions have been documented in many research papers. Misalignment in the mechanisms of the suprachiasmatic nucleus (SCN), the hypothalamic– pituitary adrenal axis (HPA), or hypothalamus interferes negatively in producing sleep disorders. Earlier animal studies showed that nicotine influences the circadian system via stimulating the nicotinic acetylcholine receptors (nAChRs) in SCN of the hypothalamus. The stimulation of nicotine to the HPA axis leads to the secretion of corticotropin-releasing hormone (CRH) in the paraventricular nucleus of the hypothalamus. This secretion stimulates the production of adrenocorticotropic hormone (ACTH) in the pituitary gland and hence leads to the increased secretion of cortisol by the adrenal gland. Sleep neurotransmitter like GABA and melatonin can be suppressed either through the inhibition of nicotine to neurons in SCN or through the stimulation of nicotine to the HPA functions by increasing the secretion of serotonin, dopamine, and noradrenaline. The purpose of this study is to investigate the relationship of tobacco and sleep disorders in Lesch typology, which is a clinical classification of four types of addicted patients to tobacco based on their level of addiction and clinical severities. Building on our previous data, we assume to see insomnia disorder in type 1 smokers who may have less total sleep time (TST), longer sleep latency, high REM sleep density, in comparison with type 2 and the control group. In this investigation, the Lesch European Smoker Classification instrument and test of biomarkers will be used to assess the tobacco abuse. To assess the sleep structure we aim to use actigraphy in combination with sleep log and polysomnography (PSG).